Thursday 25 August 2011

Disclaimer and Privacy policy

IBB is website  which functions with the sole aim to promote and spread the awareness among the people to donate blood which is helpful for the poor and needy, support child education.

Contacting Blood Donors
The users need to utilise the information provided in the site and approach the list of donors.

The details of the donors are displayed only to facilitate the users for contacting them when in need of blood in case of any emergency.

IBB  is not responsible for willingness/unwillingness to donate blood expressed by the donor listed in the site

Content
Content provided in or through the site:
  • Is provided for information purposes only.
  • Is not shared with third parties.
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  • We are not responsible for any inaccuracy in the information available on the website.
  • We are not responsible for any misuse of contact information displayed in the website. If you feel that your contact details are being misused then contact us


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Tuesday 23 August 2011

Slogans


 
[DOWNLOAD 160 SLOGANS CREATED BY ASSOCIATION OF VOLUNTARY BLOOD DONORS WEST BENGAL  ] 

Do something amazing today. Save a life. Give blood!
Give the gift that costs nothing to give.
We need each other. Please give blood.
Every drop counts. Give blood today!
It’s in your blood to help save a life.
Blood donors are special people.
From me to you—a gift of life.
Donate blood, start young.
Life is precious. Be a life-saver. Give blood!
Blood. It’s in you to give.
Accidents and medical emergencies don’t take holidays—we need blood every minute, every day of the year.
Blood costs nothing to give yet is more valuable than any other resource on earth. Don’t let the blood center run dry!
The blood you donate gives someone another chance at life. One day that someone may be a close relative, a friend, a loved one—or even you. 

Do you feel you don’t have much to offer? You have the most precious resource of all: the ability to save a life by donating blood! Help share this invaluable gift with someone in need.
Feel like you’re low on supplies? But you’re rich in the most precious resource of all: blood and the gift of life.
If you’re a blood donor, you’re a hero to someone, somewhere, who received your gracious gift of life.
Good blood helps sick people get better.
Strong communities are built on safe blood.
We need what you’ve got! Give blood.
Share life. Donate blood.
Give blood—stay healthy.
When you give blood, you’ll be doing one of the most amazing things anybody could dream of—saving a life.
Give blood today. It may be a member of your family or a friend who needs this precious resource next.
To give blood is an honor for youth…become a regular volunteer blood donor. 

Sunday 21 August 2011

Blood Disorders-Haemophilia

The body depends on clotting of blood to stop bleeding after injury and to help healing. Normal blood clotting restricts bruising after injury and stops the. bleeding into muscles and joints which can follow minor injuries associated with activities of everyday life. Normal blood clotting depends on the interaction of many substances within the blood. The critical are the clotting factors. Humans have 13 clotting factors which facilitate blood clotting whenever bleeding occurs. Haemophilia is a blood disorder where an essential clotting factor is missing, either either partly or completely. This leads to a person afflicted with this condition to bleed longer than normal. Cuts and grazes are not serious problems since such bleeding can be stopped by applying a little plaster and applying plaster/bandage. The serious issue is internal bleeding in joints, muscles and soft tissues. Haemophilia patients can experience spontaneous bleeding usually into the joints. If left untreated bleeding causes acute pain and severe joint damage leading to disability. Bleeding episodes have caused difficulties with education and employment, as well as mobility problems for many who have been crippled by the effects of regular bleeding into joints.

Haemophilia A is a type of this disorder where Clotting Factor VIII is missing. This is the classical or general type of haemophilia. Haemophilia B also called Christmas Disease is the type caused by an abnormality in the clotting protein Factor IX. Haemophilia is a genetically inherited condition. It generally affects males only but interestingly is usually carried in the female genes. However interestingly about a third of new diagnoses are where there is no previous family history. It is a lifelong condition, appears world-wide and occurs in all racial groups.

Genetic Transmission of the hemophilia gene is by females on one of their X chromosomes and may be passed to their male offspring. This is why Haemophilia is called an X-linked genetic condition. Female carriers of Haemophilia have one X chromosome with a working (normal) gene and one X chromosome with a nonworking (defective) gene. There is a 50% chance the carrier will pass the Hemophilia gene on to male offspring. In other words, there is a 50% chance that each of her male children will have Haemophilia. There also is a 50% chance the carrier will pass the Haemophilia gene on to her female offspring, meaning that there is a 50% chance each of her daughters will also be carriers.

Haemophilia occurs in about 1:5,000 male births. In general, the more severe the Haemophilia the earlier the signs of abnormal bruising or bleeding. Babies seldom have problems until they begin to crawl and start to walk. The inevitable falls and bumps which occur in toddlers may cause skin bruises and bleeding from the lips and tongue. From 2-3 years of age, bleeds (haemorrhages) may involve muscles and joints with painful swelling or a lack of movement of an arm or leg. Injections given into the muscle in the usual manner can produce a large deep bruise which may be dangerous. Immunizations are important and these may be given with safety just beneath the skin (subcutaneously). Hence early identification and control of Haemophilia is important.

Prevention and Control in this disorder is very important. As soon as a child is diagnosed parents should take precautions to prevent or reduce the occurrence of bleeding. Many of the following recommendations also apply to older people with hemophilia.
  • Injections given into the muscle in the usual manner can produce a large deep bruise which may be dangerous. Immunizations are important and these may be given with safety just beneath the skin (subcutaneously).
  • Choose sensible soft toys without sharp corners.
  • Padded clothes may be used.
  • Proper dental care is a must since infected teeth can bleed and complicate matters.
  • Encourage the child to exercise regularly and be physically fit. Strong muscles support joints and reduce the numbers of bleeds.
  • Contact sports such as boxing and rugby football are strictly prohibited.
Testing for either confirming a Haemophilia carrier or for the presence of the disorder is done in specialised labs. Early identification of a carrier helps in subsequent identification of offspring afflicted by the disorder and its control.

Treatment for Haemophilia involves replacement of the missing clotting factor. This is by injecting it on a regular basis (called prophylaxis) to help prevent bleeding, or injecting at the time a bleed occurs (called on-demand therapy). Regular treatment by prophylaxis - 2 or 3 times a week - helps the blood to clot and minimise the likelihood of long term joint damage. Unfortunately, there is no permanent way of replacing or increasing the clotting factor level.

A bleeding disorder similar to Haemophilia is called von Willebrand Disease (vWd). This disorder is normally less severe than haemophilia and can affect both sexes. When it is mild there can be a problem of regular nosebleeds, bruising, and, in women, heavy periods. There is a rare and more severe type where some of the symptoms and aspects of its treatment are similar to those of severe haemophilia.

Blood Disorders-Spherocytosis

Spherocytosis is a hereditary blood disorder. In this condition the red blood cells (RBCs) are abnormal in a spherical shape rather than the regular disc shape. The change in shape is due to a defect in one of the proteins that constitute tha outer surface of the red blood cell. As a result of the abnormal shape the RBCs are destroyed (haemolysis) at an accelerated pace inside the spleen which is the organ meant for blood filtering. The effect of accelerated destruction of the RBCs lead to complications. One, since there is inadequate haemoglobin to carry oxygen, it leads to anaemia. This can be mild with no symptoms of with severe anaemia called haemolytic anaemia. Two, the condition leads to a higher level of bilirubin in the blood which can lead to jaundice or/and lead to gallstones. Three, if there is a consequential failure of bone marrow, the patient can suffer from heart failure. However hereditary spherocytosis is generally a mild disease which does not cause many complications.

In severe cases the disorder may be detected in early childhood, or in mild cases it may go unnoticed until later in adult life. The symptoms of the condition include stomach pain, paleness or yellow color of the skin or eyes, lack of energy, lack of appetite, fever, vomiting and dark coloured urineless.

The condition is generally found in northern Europe. The condition can occur from infancy to old age. Spherocytosis can be passed from parents to their offspring. A child has 25% to 50% chance of inheriting the condition from its parent having this condition.

Families with a history of spherocytosis should have their offspring screened for this disorder. The doctor may prescribe medication, folic acid supplements and if there is an enlarged spleen, surgery may be necessary to remove the spleen. However removal of spleen is not done in children unless essential.

Blood Disorders-RH Factor

Blood Groups have a +ve or -ve appended to them. For example O+ve or O-ve. What does this +ve or -ve signify and whar relevance does it have on our health.

The +ve or -ve refers to the presence or absence of two Rhesus genes called the D or d which are inherited from each parent. A person is Rh(D) -ve if he/she has got a d gene from each parent making them d/d. A person is Rh(D) +ve if he/she has got D gene from each parent making them D/D or a D and d from the parents (either ones) making them D/d or d/D. Thus it is possible to have a Rh(D) -ve child from a father who is Rh(D)+ve and mother who is Rh(D) -ve. If the father has both a D and d gene; the offspring may inherit the d gene and shall be Rh(D)-ve if the mother is also Rh(D) -ve. The child from a D/D father and D/d or d/d mother will always be Rh(D) +ve since it will get a D from the father and either a D or a d from the mother. The d gene is not relavant. What is the presence or absence of the D which will make a person Rh(D) +ve or Rh(D) -ve.

All this is fine. The problem is that the Rh(D) +ve blood contains the D antigen which stimulates the Rh(D) -ve blood into producing antibodies against it.. A Rh(D) +ve woman would never produce an antibody against a Rh(D) -ve child, as +ve blood does not produce `anti-d` - there is no anti-Rh(d). However the problem comes where the mother is Rh(D) -ve while the child is Rh(D) +ve.

Erythroblastosis faetalis is one form of Rhesus disease. Red blood cells from the baby's blood stream may enter the blood stream of the mother across the placenta, which cause the mother to make antibodies to the baby's Rh factor. These antibodies cross the placenta and destroy the baby's red blood cells. This destruction causes the baby to become anaemic and it can die before birth. If the baby does not die before birth, its bone marrow produces extra red blood cells which are immature and releases these into the baby's blood steam. The haemoglobin from the broken down red blood cells breaks down into bilirubin, which is released into the mother's blood stream across the placenta and cleared through her metabolism. But, after the baby is born, the bilirubin builds up in it's blood stream, causing kernicterus, which is a syndrome characterised by poor feeding, poor body tone, seizures and poor breathing which may result in death. Hydrops faetalis is another form, also resulting from breakdown of red blood cells due to Rh incompatibilites, causing severe anaemia before birth. The baby is born swollen with a large liver, an enlarged heart and fluid on the lungs and in the tummy.

In such cases prevention is to be exercised. A blood test should be done at the start of pregnency to determine the D factor of the mother. If it is a Rh(D) -ve mother, further tests will be performed throughout the pregnancy to ensure that her blood is not producing Rh antibodies against her baby`s blood. If a bleed from the placenta should occur at any time during pregnancy and the foetal blood is Rh(D) positive, this would result in antibodies being produced. This is why it is essential to keep a note of when blood tests are due and what the results are.

Further she should be treated with a dose of anti-Rh antibody at about 28 weeks of pregnancy. This would help to prevent antibodies being produced if an unsuspected placental bleed were to then occur, or had already occurred within the preceding 72 hours of the injection. If this has not been done, then the mother should receive an anti-Rh gamma globulin within 72 hours of delivery. This way, the blood cells are destroyed before the three days are up and her own immune system is not provoked into producing its own anti-Rh(D).

It is highly recommended that an anti-D injection be given after any incident which could result in red Rh(D) positive cells becoming present in the mother`s bloodstream, whether this be medical intervention where Rh(D) blood has been used, a fall which may cause a placental bleed, or a miscarriage.

If the baby is born with either of the above two Rh diseases, then exchange transfusion, where equal amounts of blood are put in and taken out, is performed. Exchange transfusions can also be carried out before birth while the baby is still in the womb, if measurements of bilirubin in the amniotic fluid are becoming dangerously high.

Blood Disorders -Thalassaemia

Thalassaemia is a anaemic blood disorder. A person suffering from this disorder has defective manufacture of hemoglobin and ineffective erythropoiesis ie. manufacture of blood cells in the bone marrow. Thalassaemia is one of the most common inherited disease.This disorder is geographically common in the people of Mediterranean, African and Southeast Asian origin. The cause of the disease is not known other than the fact that it is an inherited disorder.

Thalassaemia can be ß-Thalassaemia, and alpha-thalassaemia. These can be further broken into major and minor thalassaemia.

Thalassaemia Major is the severe type where the patient has acute anaemia. The patient has frequent leg ulcers, enlarged spleen and jaundice. Ineffective erythropoiesis causes abnormal skeletal development. The long bones suffer frequent fractures and growth is stunted. Iron deposits in the heart muscles can cause complications and heart failure. Iron deposits in liver lead to dysfunctioning of the liver. The life expectancy of Thalassaemia patients is greatly diminished.

Thalassaemia Minor patients have slight anaemia but lead normal lives. Such patients at times do not even know they are carrying the disease. It is discovered only after a special blood test. Sometimes a Thalassaemia Minor patient discovers the disease after his/her child has Thalassaemia Major. Therefore it is of prime importance to know if you have Thalassaemia Minor when you want to start a family.

The only treatment for Thalassaemia Major is periodic blood transfusions every 3 to 4 weeks. Children with periodic blood transfusions lead quite normal lives upto the age of 20 to 25 years. With each blood transfusion the red cells in the new blood are broken down over a period of 4-5 months. However the iron from the red blood cells remains in the body and can damage the liver, heart and other body organs. If this is not prevented most patients die in their early twenties. Therefore the blood transfusions have to be supplemented by drug treatment to remove the extra iron from the body. The treatment is very successful and most children treated with blood transfusions and drugs lead fairly normal healthy lives. However the treatment is unpleasant and hampers with an active social life

Blood Disorders -Thrombosis

A blood clot a.k.a. as thrombus is the result of a reactions involving various proteins, their activators, and inhibitors. The process of blood clotting also involves the blood vessel wall and cellular components of blood (particularly platelets). Clotlysis (fibrinolysis) occurs via a complex series of reactions as well as a variety of tissue components. Thrombosis of a vein is a clot made up of red blood cells, white blood cells and platelets. The clot sticks to the lining of the vein and may partially or totally block the blood. Thrombosis is the basis of a number of disorders. Clots can develop anywhere in the the body, and can involve the organs.

If clotting occurs in the surface veins, it is usually not dangerous and often heals on its own. If both the surface and deep veins are involved, it may be dangerous. Heart attacks and strokes are very often due to thrombosis. The morbidity and mortality associated with clot formation is staggering. There are an estimated 5 to 20 million cases of venous thrombosis in the United States each year. Approximately 40% of the patients with deep vein thrombosis suffer pulmonary embolus which is fatal in 30% of cases. Nearly 50% of patients with deep vein thrombosis suffer sequelae lasting many years, requiring millions of dollars to treat.

Age is a risk factor in thrombosis and clotting commonly occurs in older adults. Blood clots in people younger than 40 are generally due to genetic abnormalities. Some of the inherited risk factors are abnormalities of protein C, protein S, antithrombin, and resistance to activated protein C. Clinical situations which may increase the risk of thrombosis are surgery, pregnancy, hormonal therapy, and prolonged bed rest. Each factor probably has a different risk potential for thrombosis. Once a patient reaches a certain threshold, a clot develops. Many factors remain unknown and the interaction between factors has not been clearly defined.

Blood Disorders -Leukemia

Leukemia is a cancer beginning in the bone marrow, the soft inner portion of some of your bones. In a patient affected by leukemia, the body makes too many immature or abnormal blood cells, which crowd out normal blood cells. Leukemia can spread from the bone marrow to other parts of your body, including the lymph nodes, brain, liver and spleen.

Kinds of Leukemia

There can be four main kinds of leukemia - acute or chronic leukemia that is either lymphocytic or myelogenous. In acute leukemia, the bone marrow cells remain immature. In chronic leukemia, the cells mature but are abnormal. Whether acute or chronic leukemia is lymphocytic or myelogenous depends on which bone marrow cells are affected. When the disease starts in the lymphocytes, the leukemia is lymphocytic. It is myelogenous if the granulocyte or monocyte cells of the bone marrow are affected.


Causes and Prevention

Not smoking is the most important thing you can do to help prevent leukemia. Scientists estimate that about 20 percent of adult acute leukemia cases are related to smoking. A small percentage of leukemia cases are linked to exposure to high doses of radiation (from an atomic bomb or a nuclear reactor accident, for instance) or long-term exposure to high levels of solvents such as benzene in the workplace.
Some types of leukemia are more likely to develop in people with other cancers who are treated with certain chemotherapy drugs. Combining those drugs with radiation therapy heightens the risk.
But many people with one or more of these risk factors never develop leukemia. And most of the people who do develop the disease have no risk factors at all. The cause of most leukemia is still unknown. Scientists do know that most leukemias are associated with specific gene mutations.


Symptoms and Diagnosis

Acute leukemia may cause many non-specific symptoms, including:
  • Frequent minor infections or poor healing of minor cuts
  • Swollen lymph nodes, stomach, head, arms, and gums
  • Tiny red spots in the skin Fever, headache
  • Loss of weight and/or appetite
  • Bone or joint pain
  • Difficulty in maintaining balance
  • Blurred vision
  • Easy bruising and/or bleeding
  • An enlarged, painless testicle
  • Weakness and fatigue
  • Coughing, shortness of breath
  • Seizures, vomiting
Eighty percent of patients with chronic leukemia may suffer from one of the following: weakness, fatigue, weight loss, fever, bone pain, or a feeling of fullness or pain in the abdomen, especially after eating a small meal. The remaining 20 percent have no symptoms. Their cancer is discovered by blood tests taken for other reasons or during a regular checkup.
Specific blood tests need to be carried out to determine if leukemia is the cause of symptoms. These blood tests will show changes in your blood cells. But even if these findings indicate leukemia, an accurate diagnosis usually depends on testing a sample of your bone marrow cells.
The blood and bone marrow samples are then examined under a microscope in a laboratory. These laboratory tests determine the type and subtype of leukemia to help doctors decide which treatments might work best for you.


Leukemia: Treatment

More effective ways of treating leukemia are being discovered every year. People with the disease can live for months or years, and many are now cured.
Surgery is not a cure for leukemia. Radiation therapy is sometimes used for leukemia in the central nervous system or testicles and for pain caused by bone destruction. But radiation therapy is not the primary treatment. The standard treatments for adult leukemia are chemotherapy, immunotherapy, and bone marrow transplantation.


Bone Marrow Transplantation

A bone marrow transplant, which offers some leukemia patients the best chance for a cure, is usually performed when your leukemia is in remission or when you relapse during or after treatment. The transplant enables you to receive greater amounts of chemotherapy drugs than your body could tolerate otherwise. High doses of these drugs effectively destroy cancer cells, but they also kill the normal infection-fighting cells of the bone marrow. To compensate for this destruction, high doses of anticancer drugs and of radiation therapy (to destroy any remaining cancer cells) are followed by an infusion of healthy bone marrow cells. The procedure is called an autologous transplant if these new healthy cells the body's own. If they are donated by a donor, it is known as an allogeneic transplant.
In allogeneic bone marrow transplantation, the donor's bone marrow must "match" the body's own to ensure the body is to accept the donated marrow. Relatives (usually siblings) are the people best considered as possible donors.

Divine experience




All religions talk about social responsibility, and encourage you to commit acts of charity and selfless love towards one another, as members of one family.

Giving your blood for another is an exalted act of charity and love. An act that makes you dearer to your fellow being and takes you closer to God.
Many donors have talked about a 'divine' feeling at the end of the blood donation. Instead of feeling enervated, they have felt strengthened and spiritually uplifted.
If you have any doubts, do talk to your spiritual adviser and see what he or she says. Take guidance. And act today, to feel a better human being tomorrow!

What is blood?

Blood is the red coloured fluid that flows continuously in a human being's circulatory system. Blood comprises more than 8% of the body weight of a healthy individual. On an average, every adult person has about 5 - 6 litres of blood.
The major component of blood is a fluid called plasma in which are suspended cellular elements. These are Red Blood Cells or RBC's, White Blood Cells or WBC's and tiny platelets.

Blood components, and what they do
Plasma acts as a vehicle to carry many substances like glucose, fats, and proteins, enzymes, and hormones etc., in addition to the blood cells. 

Plasma has protein components called albumin, globulin and fibrinogen. Broadly speaking, albumin maintains the structural balance of blood, globulin builds resistance to bacterial infections, fibrinogen helps blood coagulate. 

Red Cells carry oxygen from the lungs to various body tissues and take back carbon dioxide from the cells and tissues which the body gets rid of as exhaled air.
The basic substance of red cells is iron and protein, known as haemoglobin. The haemoglobin count is an indicator of the health of blood. On an average, a healthy male should have 14 - 16 gm per 100 millilitre and in a female around 12 - 14 gm per 100 millilitre. 

White cells act mainly as body scavengers and guards. They help in the immune system of the body and act as defense forces of the body, killing bacteria or any other organisms entering the body.
Platelets help in the clotting and coagulation of blood. They also repair the tiny blood vessels in the body which crack under pressure, thereby preventing haemorrhages under the skin. 

Eat the right foods for healthy blood
Blood keeps itself healthy by removing disease carrying organisms and regenerating itself, but the right nutrition plays a big role in vitality.
Blood needs an intake of 1 mg to 1.5 mg iron per day, which is met by most balanced diets. Other essential factors for red cells are present in all types of green vegetables, fruits like guava, apple, fig, and in fish, meat and eggs 

There is no SUBSTITUTE FOR BLOOD! Despite all medical advances, we have found no way of duplicating it except in our own bodies.